Best Varicose Veins Treatment in Jaipur

Dec 19, 20240 likes10 views

Varicose veins are enlarged veins near the skin’s surface, often found in the legs due to increased pressure from standing and walking. For Best Varicose Veins Treatment in Jaipur, Dr. Manish Rajput provides advanced, personalized care using modern techniques to help you achieve healthier, smoother legs and a better quality of life.

D R M A N I S H
RAJPUT
ht t ps://dr manishr ajput .com
Bookan appointment!

IN T R O D U C T IO N
Dr
. Manish Rajput is an I
nterventional
Radiologist & Team Lead, Team I
R
Jaipur
. They are the biggest team of
I
nterventional Radiologists. They are
trained from Tata Memorial Center
,
Mumbai, I
ndia. They have worked in so
many government and corporate
hospitals across the country.

Medical school (MBBS):2005-2011: -People’s
Medical College, Bhopal(MP)
DNB (Radio diagnosis):
- Apollo hospital,
Hyderabad(Telangana)
FVIR (PDCC):- Tata Memorial Centre,
Mumbai(Maharashtra)
Senior Resident: Hinduja Hospital Mumbai, SMS
Hospital Jaipur
Past Visiting Doctor:Leelavati Hospital Mumbai,
Breach Candy Hospital Mumbai, Wockhardt
Hospital Mumbai, Hinduja Hospital Mumbai
Ex Assitant Professor:JNU Medical College, Jaipur
Currently Working as Senior Consultant
Interventional Radiologist in various corporate
hospitals of Rajasthan based in Jaipur
HIS
EDUCATION

S T R E N G T H S
Ilead the biggest I
R team in the state.
Vast portfolio for I
R services.
All the team members are from Tata
Memorial Hospital, Mumbai.
Extensive experience in performing and
interpreting basic Radio-Diagnosis.
Gained experience in performing
I
nterventional Radiologic procedures.
Ipossess oratory skill by speaking at
numerous industry events.
Ability to teach complex concepts in a basic
manner
.

Varicose Veins Prostate Artery Embolization PRG
Biopsy and
fNAC
Angioplasty & Venoplasty PCN & DJ Stenting
O
U
R
S
E
R
V
I
C
E
S

+91 7729021111
dr.manish@infinityintervention.com
O-5-A, Adinath Marg, Near Surya
Hospital, C Scheme, Ashok Nagar,
Jaipur, Rajasthan 302001
C ON TA C T
US!

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ECG Interpretation and Management Introduction Electrocardiography (ECG) is a crucial diagnostic tool used to assess the electrical activity of the heart. It provides essential information about heart rate, rhythm, conduction abnormalities, myocardial ischemia, and electrolyte disturbances. Correct interpretation of an ECG requires a systematic approach and understanding of normal and pathological waveforms. Basics of ECG Interpretation 1. ECG Waves and Intervals P wave: Represents atrial depolarization. PR interval: Time from atrial depolarization to ventricular depolarization (normal: 120-200 ms). QRS complex: Ventricular depolarization (normal: <120 ms). ST segment: Represents the interval between ventricular depolarization and repolarization. T wave: Represents ventricular repolarization. QT interval: Duration of ventricular depolarization and repolarization (normal: <450 ms in males, <460 ms in females). 2. Systematic Approach to ECG Interpretation 1. Determine heart rate Regular rhythm: 300 divided by the number of large squares between R waves. Irregular rhythm: Count QRS complexes in 6 seconds and multiply by 10. 2. Assess heart rhythm Regular or irregular? Presence of P waves? Relationship between P waves and QRS complexes? 3. Evaluate cardiac axis Normal: -30 to +90 degrees. Left axis deviation: <-30 degrees (e.g., left anterior hemiblock, left ventricular hypertrophy). Right axis deviation: >+90 degrees (e.g., right ventricular hypertrophy, pulmonary embolism). 4. Analyze P wave morphology Peaked P waves (right atrial enlargement). Broad P waves (left atrial enlargement). 5. Assess PR interval Short PR: Wolff-Parkinson-White syndrome. Prolonged PR: First-degree AV block. 6. Inspect QRS complex Narrow QRS (<120 ms): Normal conduction. Wide QRS (>120 ms): Bundle branch block or ventricular origin. 7. Evaluate ST segment and T waves ST elevation: Myocardial infarction. ST depression: Ischemia or hypokalemia. Inverted T waves: Ischemia, infarction, or hypertrophy. 8. Check QT interval Prolonged QT: Risk of Torsades de Pointes. Short QT: Hypercalcemia. Common ECG Abnormalities and Management 1. Arrhythmias a) Sinus Bradycardia ECG Findings: HR < 60 bpm, normal P waves, and QRS complexes. Causes: Increased vagal tone, hypothyroidism, beta-blockers. Management: Treat underlying cause; consider atropine if symptomatic. b) Sinus Tachycardia ECG Findings: HR > 100 bpm, normal P waves, and QRS complexes. Causes: Fever, dehydration, anemia, hyperthyroidism. Management: Address underlying cause; beta-blockers if needed. c) Atrial Fibrillation ECG Findings: Irregularly irregular rhythm, absent P waves, fibrillatory waves. Causes: Hypertension, valvular heart disease, hyperthyroidism. Management: Rate control (beta-blockers, calcium channel blockers), rhythm control (amiodarone, cardioversion), anticoagulation (warfarin, DOACs). d) Atrial Flutter ECG Findings: Sawtooth flutter

HIV TREATMENT MCQ BY DR ANKUSH GOYAL.docx by Dr Ankush goyal, has 16 slides with 198 views.**HIV Treatment: A Comprehensive Overview** ## Introduction Human Immunodeficiency Virus (HIV) is a major global health challenge that affects millions of people. While there is no cure for HIV, advancements in medical research have led to effective treatment options that allow individuals with HIV to lead healthy lives. The primary treatment for HIV is antiretroviral therapy (ART), which helps to control the virus and prevent its progression to Acquired Immunodeficiency Syndrome (AIDS). This document explores the various aspects of HIV treatment, including its history, types of medications, effectiveness, side effects, challenges, and future developments. ## History of HIV Treatment The history of HIV treatment dates back to the 1980s when the first cases of AIDS were identified. Researchers quickly began searching for treatments to combat the virus. In 1987, the first antiretroviral drug, zidovudine (AZT), was approved. While it provided some benefits, AZT had significant toxicity and limited long-term efficacy. Over time, scientists developed new classes of antiretroviral drugs, leading to the combination therapy approach, which has dramatically improved patient outcomes. ## Antiretroviral Therapy (ART) ### How ART Works ART consists of a combination of drugs that target different stages of the HIV lifecycle. These medications reduce the viral load (the amount of HIV in the blood) to undetectable levels, thereby preventing the virus from weakening the immune system. ART also reduces the risk of HIV transmission to others. ### Classes of Antiretroviral Drugs There are several classes of antiretroviral drugs, each working in different ways to inhibit the replication of HIV: 1. **Nucleoside Reverse Transcriptase Inhibitors (NRTIs)** - These drugs block reverse transcriptase, an enzyme HIV uses to convert its RNA into DNA. Examples include zidovudine (AZT), lamivudine (3TC), and tenofovir (TDF). 2. **Non-Nucleoside Reverse Transcriptase Inhibitors (NNRTIs)** - NNRTIs bind directly to reverse transcriptase and inhibit its function. Examples include efavirenz and nevirapine. 3. **Protease Inhibitors (PIs)** - These drugs prevent HIV from maturing and becoming infectious by inhibiting the protease enzyme. Examples include lopinavir and atazanavir. 4. **Integrase Strand Transfer Inhibitors (INSTIs)** - These drugs prevent HIV from integrating its genetic material into human DNA. Examples include raltegravir and dolutegravir. 5. **Entry Inhibitors** - These drugs prevent HIV from entering human cells. Examples include maraviroc (CCR5 antagonist) and enfuvirtide (fusion inhibitor). 6. **Pharmacokinetic Enhancers** - These drugs improve the effectiveness of other antiretroviral medications by increasing their concentration in the bloodstream. An example is ritonavir. ## Effectiveness of HIV Treatment ### Viral Suppression When taken consistently, ART can reduce the viral load to undetectable levels. Studies have shown that individuals with u

HIV TREATMENT MCQ BY DR ANKUSH GOYAL.docxDr Ankush goyal

16 slides•198 views

**HIV Treatment: A Comprehensive Overview** ## Introduction Human Immunodeficiency Virus (HIV) is a major global health challenge that affects millions of people. While there is no cure for HIV, advancements in medical research have led to effective treatment options that allow individuals with HIV to lead healthy lives. The primary treatment for HIV is antiretroviral therapy (ART), which helps to control the virus and prevent its progression to Acquired Immunodeficiency Syndrome (AIDS). This document explores the various aspects of HIV treatment, including its history, types of medications, effectiveness, side effects, challenges, and future developments. ## History of HIV Treatment The history of HIV treatment dates back to the 1980s when the first cases of AIDS were identified. Researchers quickly began searching for treatments to combat the virus. In 1987, the first antiretroviral drug, zidovudine (AZT), was approved. While it provided some benefits, AZT had significant toxicity and limited long-term efficacy. Over time, scientists developed new classes of antiretroviral drugs, leading to the combination therapy approach, which has dramatically improved patient outcomes. ## Antiretroviral Therapy (ART) ### How ART Works ART consists of a combination of drugs that target different stages of the HIV lifecycle. These medications reduce the viral load (the amount of HIV in the blood) to undetectable levels, thereby preventing the virus from weakening the immune system. ART also reduces the risk of HIV transmission to others. ### Classes of Antiretroviral Drugs There are several classes of antiretroviral drugs, each working in different ways to inhibit the replication of HIV: 1. **Nucleoside Reverse Transcriptase Inhibitors (NRTIs)** - These drugs block reverse transcriptase, an enzyme HIV uses to convert its RNA into DNA. Examples include zidovudine (AZT), lamivudine (3TC), and tenofovir (TDF). 2. **Non-Nucleoside Reverse Transcriptase Inhibitors (NNRTIs)** - NNRTIs bind directly to reverse transcriptase and inhibit its function. Examples include efavirenz and nevirapine. 3. **Protease Inhibitors (PIs)** - These drugs prevent HIV from maturing and becoming infectious by inhibiting the protease enzyme. Examples include lopinavir and atazanavir. 4. **Integrase Strand Transfer Inhibitors (INSTIs)** - These drugs prevent HIV from integrating its genetic material into human DNA. Examples include raltegravir and dolutegravir. 5. **Entry Inhibitors** - These drugs prevent HIV from entering human cells. Examples include maraviroc (CCR5 antagonist) and enfuvirtide (fusion inhibitor). 6. **Pharmacokinetic Enhancers** - These drugs improve the effectiveness of other antiretroviral medications by increasing their concentration in the bloodstream. An example is ritonavir. ## Effectiveness of HIV Treatment ### Viral Suppression When taken consistently, ART can reduce the viral load to undetectable levels. Studies have shown that individuals with u

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Revalidation UK is a vital process ensuring healthcare professionals maintain high standards, enhancing patient safety and public trust. This mandatory assessment promotes continuous professional development, requiring practitioners to demonstrate competence, adhere to ethical guidelines, and engage in reflective practice. Despite challenges like administrative burdens and balancing clinical duties, revalidation fosters lifelong learning and accountability. Employers and regulatory bodies play a key role in supporting professionals through training, mentorship, and streamlined appraisal processes. As technology advances, revalidation UK will evolve to simplify compliance and enhance accessibility. By embracing revalidation, healthcare professionals contribute to a safer, more effective healthcare system, reinforcing trust and excellence in patient care across the UK.

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antiherpes acyclovir mcq ANKUSH GOYAL GMC PATIALA.docxDr Ankush goyal

12 slides•151 views

### **Acyclovir and Anti-Herpes Medications** **Acyclovir** is an antiviral medication used to treat infections caused by herpes viruses, including: - **Herpes simplex virus (HSV-1 & HSV-2)** – Causes cold sores and genital herpes. - **Varicella-zoster virus (VZV)** – Causes chickenpox and shingles. ### **Mechanism of Action:** Acyclovir works by inhibiting viral DNA replication. It is activated inside virus-infected cells, where it blocks viral enzymes, preventing the virus from multiplying. ### **Forms & Dosage:** - **Oral (Tablets/Capsules/Liquid)** – Used for mild to moderate infections. - **Topical (Cream/Ointment)** – For cold sores and mild skin infections. - **Intravenous (IV)** – For severe infections like herpes encephalitis. ### **Other Anti-Herpes Medications:** - **Valacyclovir (Valtrex)** – A prodrug of acyclovir with better absorption. - **Famciclovir (Famvir)** – Another antiviral with a longer duration of action. - **Penciclovir (Denavir)** – Used topically for herpes labialis (cold sores). ### **Uses:** - Treats and manages outbreaks of herpes simplex. - Reduces the severity and duration of symptoms. - Used for long-term suppression to prevent recurrent infections. - Helps in managing shingles and chickenpox. ### **Side Effects:** - Nausea, vomiting, diarrhea. - Headache, dizziness, fatigue. - Rare: Kidney issues, allergic reactions. ### **Precautions:** - Drink plenty of fluids to prevent kidney issues. - Not a cure, but helps manage symptoms. - Safe for most patients but should be used cautiously in people with kidney disease or weakened immune systems.

Topic- Biguanides, Dihydrotriazines and Miscellaneous Drugsmanashdass987

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VENTILATORS.pptx FOR NURSING STUDENTS CREATED BY KIRAN KARETHAKIRAN KARETHA

21 slides•69 views

VENTILATOR Mechanical ventilation is a form of artificial respiration that uses a breathing machine to assist patients with breathing. Mechanical ventilation is the use of a mechanical device to inflate and deflate the lungs. A mechanical ventilation is a machine that generates a controlled flow of gas into a patient’s airways. INDICATIONS Acute lung injury Apnea with respiratory arrest COPD Acute respiratory acidosis Hypoxemia hypercapnia Classification of ventilators: 1. negative pressure ventilators: mechanical ventilators exert a negative pressure on the external chest. Decreasing the intrathoracic pressure during inspiration allows air to flow into the lung, filling its volume. NEGATIVE PRESSURE VENTILATORS 1. Iron lung (drinker respirator tank) 2. body wrap and chest cuirass (tortoise shell) POSITIVE PRESSURE VENTILATORS 1. pressure cycled ventilators 2. time cycled ventilators 3. volume cycled ventilators 4. non-invasive positive pressure ventilators a) continuous positive airway pressure b) bilevel positive airway pressure

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Stress manifests in our thinking, emotions, and bodies. Recognizing those signs and building skills to address stress takes attention and effort. Cultivating skills to use your body as a tool to help you find calm and regain a sense of control can help you navigate moments of heightened anxiety. Join us on Wednesday, March 12th, to learn strategies for quieting your body and, thereby, calming your mind. Let’s Talk About It.

Urine analysis (physical, chemical and microscopic examination of urine).pdfAyat Samy

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diabetes mcq by NAME ANKUSH GOYAL (1).pdf by Dr Ankush goyal, has 14 slides with 68 views.Diabetes Mellitus: A Comprehensive Overview Introduction Diabetes mellitus is a chronic metabolic disorder characterized by hyperglycemia due to defects in insulin secretion, insulin action, or both. It affects millions of people worldwide and is a major cause of morbidity and mortality due to its associated complications. This document provides an in-depth discussion of the types, pathophysiology, clinical features, diagnosis, management, and complications of diabetes mellitus. Types of Diabetes Mellitus 1. Type 1 Diabetes Mellitus (T1DM) Autoimmune destruction of pancreatic beta cells Absolute insulin deficiency Typically presents in childhood or adolescence Requires lifelong insulin therapy 2. Type 2 Diabetes Mellitus (T2DM) Characterized by insulin resistance and relative insulin deficiency Strong genetic predisposition Associated with obesity and sedentary lifestyle Managed with lifestyle modifications, oral hypoglycemics, and sometimes insulin 3. Gestational Diabetes Mellitus (GDM) Hyperglycemia first recognized during pregnancy Increases risk of complications for both mother and baby Usually resolves postpartum but increases the risk of T2DM later in life 4. Other Specific Types Monogenic diabetes (MODY, neonatal diabetes) Secondary diabetes (due to pancreatic diseases, endocrinopathies, drug-induced, etc.) Pathophysiology Diabetes results from impaired insulin secretion, action, or both, leading to chronic hyperglycemia. The key mechanisms include: Type 1 Diabetes: Autoimmune destruction of beta cells, leading to absolute insulin deficiency. Type 2 Diabetes: Insulin resistance in peripheral tissues and inadequate compensatory insulin secretion by beta cells. GDM: Hormonal changes in pregnancy lead to insulin resistance and beta-cell dysfunction. Clinical Features Symptoms of Hyperglycemia: Polyuria (excessive urination) Polydipsia (excessive thirst) Polyphagia (excessive hunger) Unexplained weight loss Fatigue Blurred vision Complications: Acute: Diabetic ketoacidosis (DKA), hyperosmolar hyperglycemic state (HHS) Chronic: Microvascular (retinopathy, nephropathy, neuropathy) and macrovascular (coronary artery disease, stroke, peripheral artery disease) Diagnosis The diagnosis of diabetes is based on: Fasting Plasma Glucose (FPG) ≥ 126 mg/dL Random Plasma Glucose ≥ 200 mg/dL with symptoms of hyperglycemia 2-hour Plasma Glucose ≥ 200 mg/dL during an OGTT Hemoglobin A1c ≥ 6.5% Management 1. Lifestyle Modifications Healthy diet (low glycemic index, high fiber, reduced saturated fats) Regular physical activity (at least 150 minutes per week) Weight management 2. Pharmacological Therapy Oral Hypoglycemics: Metformin (first-line), sulfonylureas, DPP-4 inhibitors, SGLT2 inhibitors, thiazolidinediones Injectable Therapy: Insulin, GLP-1 receptor agonists Insulin Therapy: Required for T1DM and some cases of T2DM 3. Monitoring and Complication Prevention Regular blood glucose

diabetes mcq by NAME ANKUSH GOYAL (1).pdfDr Ankush goyal

14 slides•68 views

Diabetes Mellitus: A Comprehensive Overview Introduction Diabetes mellitus is a chronic metabolic disorder characterized by hyperglycemia due to defects in insulin secretion, insulin action, or both. It affects millions of people worldwide and is a major cause of morbidity and mortality due to its associated complications. This document provides an in-depth discussion of the types, pathophysiology, clinical features, diagnosis, management, and complications of diabetes mellitus. Types of Diabetes Mellitus 1. Type 1 Diabetes Mellitus (T1DM) Autoimmune destruction of pancreatic beta cells Absolute insulin deficiency Typically presents in childhood or adolescence Requires lifelong insulin therapy 2. Type 2 Diabetes Mellitus (T2DM) Characterized by insulin resistance and relative insulin deficiency Strong genetic predisposition Associated with obesity and sedentary lifestyle Managed with lifestyle modifications, oral hypoglycemics, and sometimes insulin 3. Gestational Diabetes Mellitus (GDM) Hyperglycemia first recognized during pregnancy Increases risk of complications for both mother and baby Usually resolves postpartum but increases the risk of T2DM later in life 4. Other Specific Types Monogenic diabetes (MODY, neonatal diabetes) Secondary diabetes (due to pancreatic diseases, endocrinopathies, drug-induced, etc.) Pathophysiology Diabetes results from impaired insulin secretion, action, or both, leading to chronic hyperglycemia. The key mechanisms include: Type 1 Diabetes: Autoimmune destruction of beta cells, leading to absolute insulin deficiency. Type 2 Diabetes: Insulin resistance in peripheral tissues and inadequate compensatory insulin secretion by beta cells. GDM: Hormonal changes in pregnancy lead to insulin resistance and beta-cell dysfunction. Clinical Features Symptoms of Hyperglycemia: Polyuria (excessive urination) Polydipsia (excessive thirst) Polyphagia (excessive hunger) Unexplained weight loss Fatigue Blurred vision Complications: Acute: Diabetic ketoacidosis (DKA), hyperosmolar hyperglycemic state (HHS) Chronic: Microvascular (retinopathy, nephropathy, neuropathy) and macrovascular (coronary artery disease, stroke, peripheral artery disease) Diagnosis The diagnosis of diabetes is based on: Fasting Plasma Glucose (FPG) ≥ 126 mg/dL Random Plasma Glucose ≥ 200 mg/dL with symptoms of hyperglycemia 2-hour Plasma Glucose ≥ 200 mg/dL during an OGTT Hemoglobin A1c ≥ 6.5% Management 1. Lifestyle Modifications Healthy diet (low glycemic index, high fiber, reduced saturated fats) Regular physical activity (at least 150 minutes per week) Weight management 2. Pharmacological Therapy Oral Hypoglycemics: Metformin (first-line), sulfonylureas, DPP-4 inhibitors, SGLT2 inhibitors, thiazolidinediones Injectable Therapy: Insulin, GLP-1 receptor agonists Insulin Therapy: Required for T1DM and some cases of T2DM 3. Monitoring and Complication Prevention Regular blood glucose